🧠 Memory Loss — GHK Perspective

Overview

In GHK, memory loss (both short-term and progressive) is understood as part of a Biological Special Program (BSP) triggered by an emotional conflict, especially in brain relays for the sensory/post-sensory cortex. The memory impairment is not seen purely as “brain tissue dying” but as a meaningful biological response to conflict, followed by healing phases.
For example, the section “(Post)Sensory Cortex Constellation” states that the manifestation is short-term memory loss when there are separation conflicts. (learninggnm.com)

Biological Conflict

Primary conflict type: A separation conflict (loss of physical contact, emotional abandonment) that impacts the sensory or post-sensory cortex.

The memory loss serves a biological purpose: “to block out the memory so that the individual is better able to cope with the separation.” (learninggnm.com)

Conflict‐active phase: The conflict registers (DHS) in the sensory/post-sensory cortex brain relay. The link to memory: when that relay is disturbed, the transmission of neural impulses is slowed (due to edema) and short‐term memory is impaired. (learninggnm.com)

Examples of conflict scenarios:

A person undergoes a sudden separation from a spouse, partner or child.
A child experiences abrupt change of residence or school, separation from friends.
An elderly person is moved to a nursing home and feels separated from familiar people or environment.

Brain-Organ Relation

The sensory and post-sensory cortex (ectodermal tissue) control the skin (epidermis), milk ducts, periosteum and memory-related transmissions. (learninggnm.com)
Memory loss in this context is tied to the relay in the brain for sensory memory — when conflict is active, the brain slows down or blocks short-term recall.
After conflict resolution, the healing phase begins and memory transmission gradually normalizes.

Phases & Symptoms

Phase	Tissue / Brain Process	Symptoms	Biological Purpose
Conflict‐active	Edema in sensory/post-sensory cortex relay → slowed synaptic transmission (learninggnm.com)	Short‐term memory loss, forgetting recent events, gaps in recall	To reduce the impact of separation conflict by blocking associated memories
Healing phase (PCL-A)	Edema reduction, cell regeneration, improved synaptic flow	Gradual memory improvement, recollection returns, possibly fatigue	The body re‐establishes full memory‐relay capacity
Post‐healing (PCL-B)	Normalized function, possible calcification of scar tissue if repeated relapses (learninggnm.com)	Memory largely restored, unless tracks of relapses persist	Completed program; stronger relay for future incident

Note on progressive memory loss: In cases where separation conflicts are repeated (tracks/relapses) or multiple constellations occur, the result may be chronic memory loss or dementia‐type patterns. (learninggnm.com)

Relationship to Diagnoses such as Dementia / Alzheimer’s

According to GHK:

What conventional medicine labels “Alzheimer’s disease” or “dementia” is interpreted as the result of repeated separation conflicts and resulting scarring in the brain relays—especially the sensory/post-sensory cortex—rather than a primary neurodegenerative disease. (learninggnm.com)
The “plaques” or brain atrophy seen in scans are considered effects of repeated conflict relapses and healing cycles — not primarily the cause. (learninggnm.com)
Elderly becoming forgetful is often linked to separation from partner/family/home, being “placed” in unfamiliar living conditions, or loss of meaningful contact. (learninggnm.com)

Case Records / Examples

In Björn Eybl’s book The Psychic Roots of Disease (10th edition) there are over 500 case examples of GNM/ GHK; memory loss is listed under the post-sensory cortex constellation. (Yes to Life)
Unfortunately, I did not identify a fully documented case available in the publicly accessible link with full details (patient demographics, time course). However, the LearningGNM PDF describes that in children the poor short-term memory may follow separation (school change, sibling birth, parental absence) and in elderly the separation may trigger a dementia‐pattern. (learninggnm.com)
Example synopsis from the text: “When the elderly start to be forgetful, it is usually regarded as the first sign of Alzheimer’s disease… According to the guidelines… the memory decline… in GNM perspective… is caused by lasting separation conflicts leading over time to dementia.” (learninggnm.com)
So while full case records (year, age, progression) are not easily extractable in accessible summary, the thematic pattern is clear.

Summary

Memory loss in GHK is interpreted as a Biological Special Program triggered by separation/conflict in the relay of the sensory/post-sensory cortex.
Short-term memory loss serves to block memory of separation-trauma so the person remains functional.
If conflicts persist or relapses occur, the condition can become chronic, mimicking dementia.
Brain changes observed (atrophy, plaques, enlarged ventricles) are framed as effects of the program not primary causes.
Sociologically, this framework shifts focus from “just aging brain” to “relational, emotional, conflict-laden life event” as root.
There are documented cases (per Eybl’s book) though full publicly-accessible datasets are limited; the theme is recurrent in GHK literature.